For the Liver

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    Sep 25, 2017 4:04 PM GMT
    How frustrating: at one time I got the idea that Larch Arabinogalactan was for the liver. Now, I have a relative with a tumor on her pancreas and her treatments have given her a liver infection. I thought: oh, Larch Arabinogalactan should support her stressed liver. So, I was about to get info about that to send to her and buy her some Ara 6 and the er4yt lit is talking about the colon. Jesus Christ.

    Just googled it before getting up to go to my library shelf for the D'Adamo books which first got me taking it. Google result (that Dr. D's product description does not include--not referencing the liver):

    It is also used to treat liver cancer, as well as a brain condition caused by liver damage (hepatic encephalopathy). Some people use it to provide dietary fiber, lower cholesterol, and to boost the immune system. - WebMD
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    Sep 25, 2017 4:06 PM GMT
    This is all Dr. D has on it:

    Help promote colon health and help support a strong immune system with ARA 6 - Pure Larch Powder.

    ARA 6 is a fiber-rich, powder derived from the Western Larch tree and is formulated for all blood types. The primary component of ARA 6 is polysaccharides (a long chain of linked sugars) called Arabinogalactans, which are high-molecular weight polysaccharides capable of up-regulating critical aspects of the immune system.

    The all-natural ingredients in ARA 6 are designed to:

    Act as a prebiotic food supply for gut microfloral balance
    Promote a balanced immune response with its adaptogenic properties
    Enhance natural liver detoxification

    ARA 6 mixes easily in liquids, which makes it ideal for children or those who may have difficulty swallowing pills.

    Dr. D'Adamo, author or Eat Right 4 Your Type, introduced larch arabinogalactan into the natural product industry over two decades ago when he wrote the first scientific review of its health effects. Unlike other larch products on the market, ARA 6 is pharmaceutical grade and of superior quality, ensuring that you're receiving the full benefit of its health enhancing properties.
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    Sep 26, 2017 1:03 AM GMT

    Blocking of organ-specific experimental metastasis

    Metastatic disease most commonly spreads to the liver, in preference to other organ sites. This has been theorized to be the result of a reaction between the galactose-based glycocongates on the metastatic cells and a hepatic-specific lectin (e.g., the D-galactose-specific hepatic binding protein) found in liver parenchyma. Several studies have compellingly shown that arabinogalactan inhibits this reaction, thus acting as a "reverse lectin."

    In one study, the effects of arabinogalactan was investigated in a syngeneic tumor-host system using a new tumor which primarily colonizes the liver upon intravenous injection. The study included systemic treatment with D-galactose and arabinogalactan as well as cell pretreatment with arabinogalactan and two other glycoconjugates. Treatment with arabinogalactan reduced the amount of liver metastases and prolonged the survival times of the animals in both studies. Host treatment was more effective than tumor cell pretreatment. This was shown to be an effect of arabinogalactan blockade of potential liver receptorsby covering of galactose-specific binding sites (29). Ths was also verified in a repeat study (30).

    In a third study, the rapid clearance and uptake by the liver of tritiated alpha 1-acid (asialo)glycoprotein from the circulation of Balb/c mice was markedly delayed after preinjection of D-galactose or arabinogalactan. The preinjection (1 h) and regular application (for 3 days after tumor cell inoculation in Balb/c mice) of the receptor blocking agents D-galactose and arabinogalactan prevented the settling of sarcoma L-1 tumor in the liver completely. Other galactans, dextrans, and phosphate-buffered saline showed no effect. Therefore, when lectins were blocked with competitive-specific glycoconjugates, colonization was prevented (31).

    Arabinogalactan completely prevented the settling of metastatic cells of sarcoma L-1 tumor in the liver of Balb/c mice and greatly reduced the colonization process of highly metastatic Esb lymphoma cells of the liver of DBA/2 mice. Therefore, when hepatic lectins were blocked with competitive glycoconjugates, tumor cell colonization of the liver could be prevented in two different model systems (32).