Sorry to welcome you to the "club." I was diagnosed in Aug '06, PSA 19.3, Gleason 9 (5 + 4). Had been misdiagnosed for 2 years so the tumor had grown out to at least the capsule. I went for radiation, radioactive seeds, and hormone-blocking therapy. I blogged my experience here on RJ: http://www.realjock.com/gayforums/150050/
(There is also an online gay prostate cancer support group on Yahoo. Very good! http://health.groups.yahoo.com/group/prostatecancerandgaymen/
... If you are still considering what treatment to pursue and have questions, I heartily recommend that you ask the guys on there. You will get lots of good answers that the doctors wont tell you. ....There are some straight men on there...they say because gays talk more openly about dicks and the sexual side effects of the disease and treatments than straight men do.)
After the hormone therapy started to wear off, my PSA shot up to .025 short of alarming per the dr. ...
... There was no more therapy to be done. You only get one shot at being cured. So I chowed down on cruciferous veggies because of their high sulfur content and ability to stimulate the liver to produce phase II enzymes that destroy cancer cells. After seven months of that, my next blood test showed an 88% drop in my PSA....lower than the accuracy of the PSA test. Now the doc says it would have to go up some 4000% for him to become concerned.
I do believe that diet can aid your body in fighting of cancer. I dont recommend relying on diet to cure yourself and not seeking medical treatment. But in conjunction with medical treatment, your diet can help your body defend itself. NOTE: dont eat or drink anti-oxidants during radiation and radioactive seed treatment, since the anti-oxidants will interfer with the radioactive ions necessary to kill the cancer cells.
Read this page on broccoli to begin to understand how cruciferous veggies can help you. http://whfoods.com/genpage.php?tname=foodspice&dbid=9
The National Cancer Institute is even studying chemicals in cruciferous veggies for their potential use as anti-cancer drugs. That's how potent they are.3,3'-Diindolylmethane
3,3-Diindolylmethane or DIM is an anticarcinogen compound derived from the digestion of indole-3-carbinol, found in Brassica vegetables such as broccoli and cauliflower. The reputation of Brassica vegetables as medicinal plants rests in part on the activities of diindolylmethane.Properties
...Because of its various potent anti-cancer properties, the National Cancer Institute of the United States has begun clinical trials of DIM as a therapeutic for numerous forms of cancer. Due to its innate immune modulating properties (potentiation of Interferon-Gamma receptors and production), DIM is also under investigation as a treatment for a variety of viral infections and antibiotic resistant bacteria. As DIM has been demonstrated to synergize with Interferon-Gamma in the potentiation of the MHC-I Complex, it is currently also under investigation as an adjuvant to IFN-G treatment models for both cancer and viral infections such as HIV, HPV and Hepatitis.Sulforaphane
Sulforaphane is an organosulfur compound that exhibits anticancer, antidiabetic, and antimicrobial properties. It is obtained from cruciferous vegetables such as broccoli. The enzyme myrosinase transforms glucoraphanin, a glucosinolate, into sulforaphane upon damage to the plant (such as from chewing). Young sprouts of broccoli and cauliflower are particularly rich in glucoraphanin.
...The anticancer activity of sulforaphane is thought to be related to the induction of phase-II enzymes of xenobiotic transformation (such as quinone reductase and glutathione S-transferase), and enhancing the transcription of tumor suppressor proteins, possibly via inhibitory effects on histone deacetylase.
Sulforaphane and diindolylmethane (another compound from Brassica vegetables) inhibit cancer growth.Isothiocyanates
Isothiocyanates, such as phenethyl isothiocyanate (PEITC) and sulforaphane, have been shown to inhibit carcinogenesis and tumorigenesis and as such are useful chemopreventive agents against the development and proliferation of cancers. They work on a variety of levels. Most notably, they have been shown to inhibit carcinogenesis through inhibition of cytochrome P450 enzymes, which oxidise compounds such as benzo[a]pyrene and other polycyclic aromatic hydrocarbons (PAHs)into more polar epoxy-diols which can then cause mutation and induce cancer development. Phenethyl isothiocyanate (PEITC) has been shown to induce apoptosis in certain cancer cell lines, and in some cases, is even able to induce apoptosis in cells that are resistant to some currently used chemotherapeutic drugs. For example, in drug resistant leukemia cells which produce the powerful apoptosis inhibitor protein BCl-2. Furthermore, isothiocyanates have been the basis of a drug in development which replaces the sulfur bonds with selenium, with far stronger potency against melanoma.