Ok, let me add a bit to this: the proposed cure is to flush out latent reservoirs of the virus so that it can be attacked with conventional HIV antiretrovirals. This is necessary, because when the virus is in a cell that is not actively producing proteins, it is not susceptible to any of these drugs. This strategy has been proposed and tied for at least a decade, with little success. This is just the latest installment of of that approach; the use of histone deacetylase inhibitors to turn on transcription rather unspecifically in a wide range of cells, in the hopes that enough of the reservoir cells will be among them. It is currently being tried in multiple clinical studies. In fact, the Danish group mentioned in the article is just going into phase I and seems to be a little late to the game. I believe there are already phase III studies in progress elsewhere. It would of course be great if this worked, but my suspicion is that it'll be only marginally effective - some reservoir cells will be flushed out, but in most cases, enough will remain so that the virus will come back once the antiretrovirals are discontinued. But it might give us some insight into how to turn these cells on, and the next step would be better targeting of just the reservoir cells, instead of this broad paintbrush approach.